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6. 2007Äê-2008Ä꣬Ö÷Òª²ÎÓë¹ú¼Ò×ÔÈ»¿ÆÑ§»ù½ðÃæÉÏÏîÄ¿(**)£º¡°¼Ò×åÐÔ·¿²üÖ²¡»ùÒòµÄʶ±ð¡±¡£¸ÃÏîÄ¿ÊÇͬ¼Ã´óѧ³ÂÒ庺½ÌÊڵĿÎÌ⣬±¾È˲©Ê¿ÆÚ¼ä¿ÎÌⲿ·ÖÄÚÈÝÓë´ËÓйØ
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2£® ºéÀö£¬Áõ½ðÇÍõÓ¨çù£¬ÅËÏþ½Ü£¬ÏÄÔÆÁú£¬ÕÅÊ÷Áú£¬¸ßÁ¬¾ý£¬ÑîÑÓ×Ú. ³¤QT13¼ÒϵµÄÁÙ´²±íÏÖºÍÐĵçÑ§ÌØÕ÷Ñо¿¡£ÖлªÐÄÂÉʧ³£Ñ§ÔÓÖ¾£¬2014£¬18£¨4£©£º269-274.(Áõ½ðÇïΪͨѶ×÷Õß)
3£® Wang F, Liu J, Hong L, Liang B, Graff C, Yang Y, Christiansen M, Olesen,S?ren-Peter, Zhang L, Kanters J?rgen K.. The phenotype characteristics of type 13long QT syndrome with mutation in KCNJ5 (Kir3.5-G387R). Heart Rhythm,2013,10:1500-1506. £¨IF£º5.101£©
4£® Yang Y, Yang Y,Liang B, Liu J, Li J, GrunnetM, Olesen P, Rasmussen HB, Ellinor PT, Gao LJ, Lin XP, Li L, Wang L, Xiao JJ,Liu Yi, Liu Ying, Zhang SH-L, Liang D, Peng LY, Jespersen T, Chen YH.. Identification of a Kir3.4 mutation in congenital long QT syndrome. Am J Hum Genet,2010,(86):827~880£¨IF£º12.3£©
5£® Yang Y, Li J, Lin X, Yang Y, Hong K, Wang L, Liu J, Li L, Yan D, Liang D, Xiao J,Jin H, Wu J, Zhang Y, Chen YH. Novel KCNA5 loss-of-functionmutations responsible for atrial fibrillation. J Hum Genet. 2009, 54(5):277-83.£¨IF£º12.3£©
6£® Áõ½ðÇï, ÁõÓ¨, ¸ßÁ¬¾ý, µÈ. ¼Ò×åÐÔµçÎÉÂÒÐÔÐÄÔಡδֲÈëICD»¼Õߵij¤ÆÚËæ·ÃÑо¿ ÖлªÐÄÂÉʧ³£Ñ§ÔÓÖ¾2008, 12£¨2£©£º107-109.
7£®Fouts K, Fernandes B, Mal N, Liu J, Laurita KR. Electrophysiologicalconsequence of skeletal myofibroblast transplantation in normal and infarctedcanine myocardium. Heart Rhythm. 2006, 3(4):452-61.£¨IF£º4.444£©
8£®Liu Jinqiu, Laurita KR. The mechanism ofpause-induced Torsade de Pointes in LQT syndrome. JCE, 2005, 9: 981-987. £¨IF£º3.798£©
9£®Áõ½ðÇÁÖÙ»£¬ÁõÉÙÎÈ£¬ÑîÑÓ×Ú£¬ÁÖÖκþ. ²»Ó¦ÆÚÀëÉ¢ÓëÐĵ粻Îȶ¨ÐÔ¹ØÏµµÄ¼ÆËã»úÄ£ÄâÑо¿. ÖлªÐÄÂÉʧ³£Ñ§ÔÓÖ¾, 2001£¬£¨2£©£º106-109.
10£® Liu Jinqiu, Wang Yingqi, Xia Yunlong, LiShijun, Yang Yanzong. Clinical investigation of Seven cases of idiopathic longQT syndrome from the same family pedigree. PACE, 2001,24:s66.(IF:1.25)


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